Publications

Rühl, N., Lopez-Rodriguez, E., Albert, K., Smith, B.J., Weaver, T.E., Ochs, M., Knudsen, L.: Int J Mol Sci. 2019;20(17). To access the full article, click here: High surface tension at the alveolar air-liquid interface is a typical feature of acute and chronic lung injury. However, the

Kloth, C., Gruben, N., Ochs, M., Knudsen, L., Lopez-Rodriguez, E.: Am J Physiol-Lung C. 2019;317(1):L109-26. To access the full article, click here: Bleomycin-induced lung injury and fibrosis is a well-described model to investigate lung inflammatory and remodeling mechanisms. Rat models are clinically relevant and are also widely

Beike, L., Wrede, C., Hegermann, J., Lopez-Rodriguez, E., Kloth, C., Gauldie, J., Kolb, M., Maus, U.A., Ochs, M., Knudsen, L.: Lab Invest. 2019;99(6):830-52. To access the full article, click here: In human idiopathic pulmonary fibrosis (IPF), collapse of distal airspaces occurs in areas of the lung

Hollenbach, J., Lopez-Rodriguez, E., Mühlfeld, C., Schipke, J.: Int J Mol Sci. 2019;20(10). To access the full article, click here: Diabetes and respiratory diseases are frequently comorbid conditions. However, the mechanistic links between hyperglycemia and lung dysfunction are not entirely understood. This study examined the effects of high

Lopez-Rodriguez, E., Roldan, N., Garcia-Alvarez, B., Pérez-Gil, J.: J Lipid Res. 2019;60(2):430-5. To access the full article, click here: TLC has traditionally been used to analyze lipids isolated from membrane complexes. Here, we describe a method based on the combination of TLC and SDS-PAGE to qualitatively analyze

Beneke, V., Küster, F., Neehus, A.L., Hesse, C., Lopez-Rodriguez, E., Haake, K., Rafiei Hashtchin, A., Schott, J.W., Walter, D., Braun, A., Wolkers, W.F., Ackermann, M., Lachmann, N.: Sci Rep. 2018;8(1):16281. To access the full article, click here: Macrophages are key cells of the innate

Lugones, Y., Blanco, O., Lopez-Rodriguez, E., Echaide, M., Cruz, A., Pérez-Gil, J.: PLoS ONE. 2018;13(9):e0204050. To access the full article, click here: Inactivation of pulmonary surfactant by different components such as serum, cholesterol or meconium contributes to severe respiratory pathologies through destabilization and collapse of airspaces. Recent

Mucci, A., Lopez-Rodriguez, E., Hetzel, M., Liu, S., Suzuki, T., Happle, C., Ackermann, M., Kempf, H., Hillje, R., Kunkiel, J., Janosz, E., Brennig, S., Glage, S., Bankstahl, J.P., Dettmer, S., Rodt, T., Gohring, G., Trapnell, B., Hansen, G., Trapnell, C., Knudsen, L., Lachmann, N., Moritz, T.: Stem Cell Reports.

Knudsen, L., Lopez-Rodriguez, E., Berndt, L., Steffen, L., Ruppert, C., Bates, J.H.T., Ochs, M., Smith, B.J.: Am J Resp Cell Mol. 2018;59(6):757-69. To access the full article, click here: Lung injury results in intratidal alveolar recruitment and derecruitment and alveolar collapse, creating stress concentrators